Case Study Of Acute Pulmonary edema Discussion

Questions:

1.Refer to the case scenario provided below for Ms Foley who has been diagnosed with acute pulmonary edema. From the assessment information in the scenario, you are to analyse the information provided and then complete two (2) charts – Observation Chart and Fluid Balance Chart (FBC).

2.Explain the inter-professional model of care required for Ms Foley while in hospital. You are to include the most appropriate national nursing regulatory standards linked to Ms Foley’s care. A starting point is to read the Registered Nurse Standards for Practice (NMBA 2016)Case Study Of Acute Pulmonary edema Discussion.

This assignment is about a patient named Ms. Martha Foley, a 35 year old lady who is suffering from acute pulmonary edema and was admitted in the hospital prior to 5 days because of post multi-trauma caused by a car accident. She is also suffering from multiple fractures in both legs and smoke inhalation as being trapped in car for 15 minutes.

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This paper has been divided into two parts. Part 1 contains the description of Ms. Foley and her history along with the treatment in the hospital and the analysis of the Observation Chart and Fluid Balance Chart. It also contains a brief description of acute pulmonary edema, its pathophysiology and the implications of nursing assessment of this situation.

Part 2 contains the inter-professional model of care required for Ms Foley while in hospital.

Ms. Foley is a full time worker at a supermarket and is a victim of domestic violence from her former partner. She had a good relationship and support from her friends and co-workers. The day of the accident she and her friend Ms. Annie Jones were travelling in the same car. Ms. Jones was also admitted in the same hospital. Ms. Foley was a smoker of 10 cigarettes per day and also intake drugs such as methamphetamines IV and cocaine. But don’t drink alcohol and is a vegetarian. She had suffered from left sided heart failure in the past and is treated by cardiomyopathy.

Fluid balance chart describes that Ms. Foley is given compound Sodium acetate was given intravenously from 01:00 am to 08:00 am at 200mls per hour and only 100mls is drained out through urine. It has been shown in appendix 1. Case Study Of Acute Pulmonary edema Discussion

Observation chart describes about the general physical conditions of Ms. Foley during the time of her admission in the hospital. The following things were measured:

  • Respiratory rate – 16-20 breathe/min
  • O2 Saturation – 95-97%
  • Air delivery method – RA
  • Blood pressure – 120/40-150/80 mmHg
  • Pulse rate – 70-90 beats /min
  • Temperature – 36.6-37.0 ?
  • Consciousness/Sedation – 0
  • Pain score – 0-5

Acute pulmonary edema is a type of disease in which the alveoli gets filled up with fluid and the person is unable to breathe. As the fluid gets accumulated creates misbalance in exchange of gases and leads to dyspnea and hypoxia. Acute pulmonary edema (APO) is caused by two main mechanisms. The first mechanism is elevations in pulmonary pressures due to severe increase in intra-cardiac pressure of chambers and second is severe lung injury causing increase in pulmonary vascular permeability (Liesching et al. 2014).

Pulmonary edema caused due to lung injury is termed as acute respiratory distress syndrome (ARDS) while pulmonary edema which is due to acute elevations in pulmonary micro vascular pressures is termed acute cardiogenic pulmonary edema (Lavin et al. 2015)Case Study Of Acute Pulmonary edema Discussion.

The pathophysiology of these two types of edema is almost different due to which their treatments are also different. This document relates specifically to acute cardiogenic pulmonary edema (Zeiss 2016).

Acute cardiogenic pulmonary edema is caused by an intense ischemic occasion bringing about cardiovascular brokenness. Intense diastolic disappointment may likewise be caused from hypertension and atherosclerosis. Less generally the intense sight might be optional to other essential occasions, for example, intense valvular issue or arrhythmia (Elias et al. 2015)Case Study Of Acute Pulmonary Oedema Discussion.

The acute cardiac dysfunction that happens from a background marked by myocardial ischemia brings about expanded cardiovascular end diastolic weights which thus result in hoisted pneumonic micro vascular weights and the improvement of intense cardiogenic aspiratory oedema (Vlaar and Juffermans 2013). There is a decrease in cardiovascular yield with ensuing solid enactment of the thoughtful sensory system and the renin-angiotensin framework. This outcomes in lifted blood weights and expanded delayed consequences on the heart starting a gooey cycle of expanded myocardial oxygen request and further lessening in cardiovascular yield (Luks et al. 2014)Case Study Of Acute Pulmonary edema Discussion.

In the problems of heart failure there are homeostatic systems that prompt liquid maintenance and pneumonic blockage. In APO there might be starting of liquid fill up, however the essential pathophysiology won’t be reliant on the benchmark liquid status. It is significant that s number of these patients is hemoconcentrated on introduction because of the gathering of up to 2 litres of liquid in their lungs (Spoletini et al. 2015). They may then show up hemodiluted 24 hours after the fact when they are in the recuperation stage. As it were they give off an impression of being enduring a more prominent plasma volume as of now, which is suggestive that the issue is not fundamentally a liquid over-burden issue. By and large of APO the issue is less liquid abundance; rather it is an instance of liquid in the wrong “compartment”, (intra-alveolar, as opposed to intra-vascular) (Duffy et al. 2015).

The nursing care plan depends upon the appearance of the patient’s respiratory status. To perform the assessment of nursing care for acute pulmonary edema the nurse must observe the individual’s responses (Akoumianaki et al. 2014)Case Study Of Acute Pulmonary edema Discussion.

  1. To assess the respiratory rate, intensity, and effort, including nasal flaring, and abnormal patterns of breathe.
  2. To assess the lungs to locate the areas of decreased ventilation.
  3. To monitor the mental status of the patient for beginning of agitation, confusion, and extreme tiredness.
  4. To monitor the symptoms of atelectasis: bronchial or tubular sounds of breathing, crackles, reduced chest congestion, and shift of trachea to the affected side.
  5. To monitor the alteration in Blood Pressure.
  6. To observe the nail beds, skin cyanosis and mucous membranes of mouth.
  7. To monitor oxygen saturation continuously, using pulse oximeter.
  8. Monitor the effects changes in position during oxygenation (ABGs), venous oxygen saturation and pulse oximetry.
  9. To assess the ability of patient to excrete out mucous by coughing. Take note of the quantity, colour, and consistency of the sputum.
  10. Evaluate the patient’s hydration status.
  11. Check on Hgb levels.
  12. Monitor chest x-ray reports Case Study Of Acute Pulmonary edema Discussion

The following are the therapeutic nursing interventions for acute pulmonary edema:

  1. A nurse must check the patient’s position regularly so that she must not fall from bed.
  2. The nurse must turn the patient after every 2 hours and monitor the venous oxygen diffusion after turning. If it drops below 10% or fails to return to baseline promptly, turn the patient back into a supine position and evaluate oxygen status.
  3. If patient is suffering from obesity then change the patient to Trendelenburg position at 45 degrees which can be tolerated by the patient.
  4. If the patient is extremely dyspnoeic, try the patient to lean over a bedside table, if it is tolerable by patient.
  5. Maintain the oxygen delivering device as per the instruction, which can maintain oxygen saturation.
  6. Avoid increasing the concentration of oxygen to the patients with COPD.
  7. Encourage or assist with ambulation as per physician’s order.
  8. Help the patient to take deep breath and control coughing. To support the patient to inhale deeply, then holding the breath for some seconds, and cough for two to three times by keeping the mouth open.
  9. Encourage slow deep breathing using an incentive spirometer as indicated.
  10. For postoperative patients, assist with splinting the chest. Case Study Of Acute Pulmonary edema Discussion

The initial management of patients with cardiogenic pulmonary edema (CPE) should address the ABCs of resuscitation, that is, airway, breathing, and circulation. Oxygen should be administered to all patients to keep oxygen saturation at greater than 90%. Any associated arrhythmia or MI should be treated appropriately (Lumb 2016).

Methods of oxygen delivery incorporate the utilization of a face covering, non-intrusive pressure support ventilation (which incorporates bi-level positive aviation route weight [BiPAP] and consistent positive airway pressure [CPAP]), and intubation and mechanical ventilation (Stocker et al. 2014). The method that will be utilized relies upon the proximity of hypoxemia and acidosis and on the patient’s level of cognizance. For instance, intubation and mechanical ventilation may end up noticeably important in instances of tireless hypoxemia, acidosis, or changed mental status. The utilization of non-intrusive weight bolster ventilation in acidotic patients with serious intense cardiogenic aspiratory oedema does not give off an impression of being related with unfavourable results (early mortality and intubation rates) in these patients (Adam, Osborne and Welch 2017)Case Study Of Acute Pulmonary Oedema Discussion.

Intravenous:

Initial dose: Frusamide is given intravenously and the dosage is 20-40 mg (slowly for 1 to 2 minutes) or may increase the dosage by 20 mg later by 2 hours after the previous dose until the desired diuretic effect has been obtained.

Use: Treatment of oedema associated with congestive heart failure, cirrhosis of the liver, and renal disease, including the nephrotic syndrome, especially when an agent with greater diuretic potential is desired (Soni et al. 2017)Case Study Of Acute Pulmonary Oedema Discussion.

Oral:

Initial dose: 20 to 80 mg orally once; may repeat with the same dose or increase by 20 or 40 mg no sooner than 6 to 8 hours after the previous dose until the desired diuretic effect has been obtained.

Maintenance dose: Administer the dose that provided the desired diuretic effect once or twice a day.

Maximum dose: 600 mg/day in patients with clinically severe oedematous states.

Comments:

-Oedema may be most efficiently and safely mobilized by giving this drug on 2 to 4 consecutive days each week.

-When doses greater than 80 mg/day are given for prolonged periods of time, careful clinical observation and laboratory monitoring are particularly advisable.

Use: Treatment of oedema associated with congestive heart failure, cirrhosis of the liver and renal disease, including the nephrotic syndrome, especially when an agent with greater diuretic potential is desired.

The Inter-Professional Model of Patient Care (IPMPC©) was introduced in 2006 when inter-professional collaboration started as a priority of policy agenda in Canada. This model causes the experts to enhance better care the patients and their families which likewise can manage the extra issues of human medicinal services. The models speak to a pedantic program, a group based affair and a between proficient recreation encounter. The instructional program stresses between proficient group building abilities, information of callings, tolerant focused care, benefit taking in, the effect of culture on medicinal services conveyance and a between proficient clinical segment (Maggiore et al. 2014)Case Study Of Acute Pulmonary Oedema Discussion.

Nurses were ready for Ms Foley to attend her while she was lying flat on her bed, trying to sit up, and gasping for air. She is worried, stating she is ‘scared’, and that she has a weird feeling that she can only describe as ‘a sense of impending doom’. Vital signs taken and are the following: Pulse 120bpm and regular, BP 100/50, RR 34, SaO2 92% on RA. Her lips have started to turn blue and the nail beds on her fingers are cyanotic. She remains dyspnoeic and desperate for air. She is coughing.

The integral part of inter-professional care of nursing is quality improvement. ACHS is a handbook which gives information of management of risks and improvement of quality to assist organisations and to manage the risks at the organisational levels and to ensure that quality of care and services are integrated (Powell et al. 2016).

PE teams have found their place in health care. Teams do not replace the physician-patient relationship, but rather enhance it—creating a more comprehensive, efficient, and tailored health care experience (Short et al. 2014)Case Study Of Acute Pulmonary Oedema Discussion.

The care convention was created by the IP group amid a CPE workshop and encouraged group gatherings. It was utilized as the guide by medical caretakers or doctors associates who gave mind at the visits. Components of the IP mind convention included visual signs, for example, publications on the exam room dividers that represent the body frameworks affected by diabetes, and an envelope with data, worksheets and apparatuses created by the IP group on parts of connecting with relatives for help, eating regimen, exercise and medical administration.

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Conclusion

Thus through the above discussion it can be concluded that Ms. Martha Foley had an accident and detected by acute pulmonary oedema which is a type of disease in which the lungs is filled up with fluid. At first in this task there had been included clinical assessment tools; pathophysiology and its applications; nursing care plan; nursing interventions; pharmacological uses of frusamide; inter-professional model of care, its risk management and its implications Case Study Of Acute Pulmonary edema Discussion